therapy. Thus, Lazarus (1986) notes that “terms such as ‘expectancies,’ ‘encoding,’ ‘plans,’ ‘values,’ and ‘self- regulatory systems,’ all operationally defined, have crept into the behavior literature” (p. 251). More recently, it has shown that versions of behavior modification are effective in treating major depression (Whooley and Simon 2000; Hollon, Thase, and Markowitz 2002). However, pharmacological treatment is superior in patients with more severe depression (Schulberg et al. 1998).

In the last decades of the twentieth century, research on psychotherapy has focused on which forms of therapy are effective and just what specific disorders they are effective for. Chambless et al. (1998) have reviewed this research and publish in their paper a listing of therapies that have been shown to be either “well established” or “probably efficacious” based on empirical research. Therapies in both these categories are almost exclusively of the behavioral or cognitive behavior type.

The early behavior modifiers made the unfounded claim that all disordered behaviors were the result of learning or conditioning of one type or another. While this rather grandiose claim was wrong—for example, many behavioral disorders are biochemically caused—other disorders were properly thought to be due to learning factors. Behavior therapies were quite effective at treating these. Examples include phobias (Paul 1969a, 1969b), certain specific types of depression (Rehm 1981), and other disorders ranging from obsessive-compulsive disorders to some sexual disorders (Bandura 1969).

Interestingly, the advent of behavioral modification techniques that focus on the disordered behavior itself, rather than on hypothetical psychological causes such as unresolved Oedipal complexes and the like, provided an opportunity to test one strong prediction made by psychoanalytic theory. According to psychoanalytic theory, the overt disordered behavior a patient displays is merely a symptom of some hidden, deep psychological cause According to psychoanalytic theory, it would not be sufficient to simply eliminate the symptom (i.e., the behavior), because the underlying cause of the disorder would still be there and would only cause some other problem behavior (i.e., symptom) in the future. This is known as the symptom substitution hypothesis. Studies of patients who have been treated behaviorally have shown no evidence of symptom substitution (Bandura 1969; Franks 1969).

The evidence indicates that the belief that psychotherapies other than behaviorally oriented ones actually work is a myth. Another myth held dear by psychologists is what Dawes (1994) terms the “myth of expertise.” This is the deeply held belief that clinical judgments based on interviews and personal interaction with patients or clients results in better judgments about such variables as dangerousness, psychopathology, neuropsychological status, and so on than do judgments based only on the results of valid and reliable empirical tests. It has been known since the early 1950s that this belief is simply wrong. Not only are judgments based on what is often termed “clinical experience” no better than those based purely on statistical calculations, they are almost always worse (Meehl 1954; Faust and Ziskin 1988; Dawes, Faust, and Meehl 1989; Dawes 1994; Ziskin 1995). Clinicians continue to believe that their experience-based judgments are superior because of effects such as selective memory: They are much more likely to remember the instances in which their judgments happened to be correct than those in which they were not. This is precisely the sort of cognitive illusion that supports beliefs in other invalid belief systems such as ESP (see chapter 2) and astrology, moon madness, and biorhythm theory (see chapter 6). The realization of the “myth of expertise” has led some (e.g., Dawes 1994; Hagen 1997) to question whether testimony by psychologists and psychiatrists based on interviews, projective tests, and other invalid techniques is anything other than junk science.

NEUROBIOLOGY AND MENTAL DISORDERS

Although Freud believed that the constructs in psychoanalytic theory were biologically real, he also believed that the causes of psychological disorders, serious and minor, could be traced to patients’ experiences while growing up. The early years, during which psychosexual development was said to take place, were especially important. If the child was exposed to aberrant situations during this period, disorders of psychosexual development could occur that would appear in adulthood. Thus, for example, overt male homosexuality was thought to be due to the boy’s failure to form a normal identification with his father, due to either a cold, unloving father or a domineering mother. Mothers, in psychoanalytic thought, are often responsible for the psychological disturbances of their children. Depression is said to be caused when a loss during adulthood reactivates the represssed feelings of the traumatic loss during childhood of the mother’s affection. Psychoanalytic psychologist Bruno Bettelheim (1967) argued that the childhood disorder autism is caused by inept, unloving, and cold mothers, a now totally discredited view.

Within the past decade great strides have been made in understanding the nature of many disorders. In a host of such disorders that were previously thought to be “psychological”—that is, caused by some abnormality in the interactions the individual had with his parents or peers, usually as a child—the actual causes have been determined to be physiological, usually involving abnormalities in the chemistry of the brain. Considerable evidence now exists to show that human homosexuality is caused, at least in large part, by hormonal influences that take place while the brain is developing in utero (Ellis and Ames 1987; Goy and McEwen 1980), although specific cultural influences are also at work (Green 1987; Money 1987). There is also evidence that there are anatomical differences between the brains of straight and gay human males (LeVay 1993).The important fact here is that modern research on the etiology of homosexuality fails to support the psychoanalytic view.

To take another example, there are several types of depression. One type, called reactive depression, is a reaction to some environmental event, such as the loss of a friend, lover, or parent, or even the loss of an environment, as in the homesickness one sometimes sees in first-year college students. Research on this type of depression shows no support for the psychoanalytic “explanation.” More serious types of depression are biological in nature. So-called endogenous depression is caused by abnormalities in the levels of certain neurotransmitters, chemicals that allow neurons to transmit information to one another, in the brain (see Rosenzweig, Leiman, and Breedlove 1999, for a brief review). Endogenous depression is not linked to any objectively depressing event in the patient’s environment and can be treated, although not perfectly, with medications designed to normalize the patient’s brain chemistry. An even more serious type of depression is seen in manic-depressive psychosis, where the patient alternates between periods of deep depression and high-energy mania. Chemical treatment is available for this disorder, although it is sometimes necessary to use electroshock treatment. It was long thought that there was only one type of manic-depressive psychosis. Recent genetic studies, however, have revealed two separate chromosomal locations at which genes that can cause manic-depressive psychosis are found (Egeland et al. 1987; Hodgkinson et al. 1987), showing that there are two genetically distinct versions of the disorder. Such findings provide a far more profound understanding of depression than psychoanalytic cliches. Even suicidal behavior may be understood in terms of an underlying neurochemical abnormality (Mann and Stanley 1986) such that suicide-prone individuals may differ from others in levels of certain neurotransmitters in the brain.

Childhood or infantile autism is a serious developmental disorder in which the child’s language develops poorly and the child ignores her surroundings and engages in stereotyped “self-stimulatory” behaviors such as waving the hands back and forth in front of the face for extended periods of time. The autistic child may engage in self-damaging behavior such as head banging and the chewing of her own flesh. It seems hard to imagine that such a severe disorder could be caused simply by cold, inept parents, as Betteiheim (1967) has contended. Bettelheim’s “blame the mother” approach no doubt held up research into the actual causes of autism for decades. It now seems likely that autism is not a single disorder but a group of disorders with different causes that may run the gamut from subtle brain damage before birth (Patzer and Volknaer 1999) to food allergies (Seroussi 2000). In addition to blocking research, Bettelheim caused an untold amount of grief for parents, especially mothers, who believed that they had caused their chid’s autism (Gardner 2000; Dolnick 1998).

Other disorders often thought to be psychological are now known to be caused by neurochemical abnormalities. One of the best-known is schizophrenia, which Freud believed was related to narcissism or self-love. In fact, schizophrenia (there are probably at least two types) is now known to be a genetically determined neurochemical disorder in which environmental influences such as stress may play some, but not a major, role (Rosenzweig, Leiman, and Breedlove 1999). Another disorder now linked to underlying biochemical abnormalities is

Добавить отзыв
ВСЕ ОТЗЫВЫ О КНИГЕ В ИЗБРАННОЕ

0

Вы можете отметить интересные вам фрагменты текста, которые будут доступны по уникальной ссылке в адресной строке браузера.

Отметить Добавить цитату